Diferencia entre revisiones de «Template:Ischemic Stroke Cause»
Sin resumen de edición |
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| Línea 2: | Línea 2: | ||
*Thrombotic (80% of ischemic CVA) | *Thrombotic (80% of ischemic CVA) | ||
**Atherosclerosis | **Atherosclerosis | ||
**Vasculitis | **[[Vasculitis]] | ||
**[[Vertebral and carotid artery dissection]] | **[[Vertebral and carotid artery dissection]] | ||
***Often preceded by yoga, spinal manipulation, coughing, vomiting | ***Often preceded by yoga, spinal manipulation, coughing, vomiting | ||
**Polycythemia | **[[Polycythemia]] | ||
**Hypercoagulable state (oral contraceptives, antiphospholipid antibodies, protein S and C deficiencies, sickle cell anemia) | **Hypercoagulable state (oral contraceptives, antiphospholipid antibodies, protein S and C deficiencies, sickle cell anemia) | ||
**Infection | **Infection | ||
** | **[[Toxicologic exposure]] ([[cocaine]], [[amphetamines]], etc.) | ||
*Embolic (20% of ischemic CVA) | *Embolic (20% of ischemic CVA) | ||
**Valvular vegetations | **[[endocarditis|Valvular vegetations]] | ||
**Mural thrombi | **Mural thrombi | ||
**Arterial-arterial emboli from proximal source (ex. ''amaurosis fugax'' -> emboli from a proximal carotid artery plaque embolizes to the | **Arterial-arterial emboli from proximal source (ex. ''[[amaurosis fugax]]'' -> emboli from a proximal carotid artery plaque embolizes to the ophthalmic artery, causing transient monocular blindness) | ||
**[[Fat embolism syndrome|Fat emboli]] | **[[Fat embolism syndrome|Fat emboli]] | ||
**Septic emboli | **Septic emboli | ||
*Hypoperfusion | *[[Shock|Hypoperfusion]] | ||
**Cardiac failure resulting in systemic hypotension | **Cardiac failure resulting in systemic hypotension | ||
Revisión del 15:04 31 ago 2019
Ischemic stroke causes (87% of all strokes)
- Thrombotic (80% of ischemic CVA)
- Atherosclerosis
- Vasculitis
- Vertebral and carotid artery dissection
- Often preceded by yoga, spinal manipulation, coughing, vomiting
- Polycythemia
- Hypercoagulable state (oral contraceptives, antiphospholipid antibodies, protein S and C deficiencies, sickle cell anemia)
- Infection
- Toxicologic exposure (cocaine, amphetamines, etc.)
- Embolic (20% of ischemic CVA)
- Valvular vegetations
- Mural thrombi
- Arterial-arterial emboli from proximal source (ex. amaurosis fugax -> emboli from a proximal carotid artery plaque embolizes to the ophthalmic artery, causing transient monocular blindness)
- Fat emboli
- Septic emboli
- Hypoperfusion
- Cardiac failure resulting in systemic hypotension