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==Background==
==Background==
*Due to trauma to temporoparietal area w/ associated skull fx and meningeal artery damage
*Bleeding between the skull and dura mater, typically from rupture of the middle meningeal artery
*Classic presentation of LOC > lucid interval > LOC only occurs in 20%
*Usually associated with temporal bone fracture<ref name="bullock">Bullock MR, et al. Surgical management of acute epidural hematomas. ''Neurosurgery''. 2006;58(3 Suppl):S7-15. PMID 16710967.</ref>
*Injury to brain is often absent so good recovery if hematoma evacuated in time
*Accounts for 1-4% of traumatic [[Head trauma (main)|head injuries]]
*Bimodal age distribution: most common in adolescents and young adults
*Rare in elderly (dura more adherent to skull) and infants <2 years
*Carries ~5-10% mortality with prompt treatment; higher if uncal herniation occurs


==Diagnosis==
==Clinical Features==
*Classic presentation (lucid interval in ~30% of cases):
**Initial loss of consciousness (LOC) after head trauma
**Transient period of lucidity
**Rapid deterioration with decreasing [[GCS]], ipsilateral pupil dilation
*Headache, nausea, vomiting
*Signs of [[Increased intracranial pressure|increased ICP]]: [[Cushing response]] (hypertension, bradycardia, irregular respirations)
*Ipsilateral fixed, dilated pupil ([[Uncal herniation|uncal herniation]])
*Contralateral hemiparesis
*May present without lucid interval, especially with severe injury


==Differential Diagnosis==
==Differential Diagnosis==
{{Intracranial hemorrhage DDX}}
*[[Subdural hemorrhage]]
*[[Subarachnoid hemorrhage]]
*[[Intracerebral hemorrhage]]
*[[Cerebral contusion]]
*[[Diffuse axonal injury]]
 
==Evaluation==
*Non-contrast CT head — test of choice<ref name="dent">Dent DL, et al. Prognostic factors after acute subdural hematoma. ''J Trauma''. 1995;39(1):36-43. PMID 7636907.</ref>
**Biconvex (lens-shaped) hyperdense collection
**Does NOT cross suture lines (confined by dural attachments)
**May see associated skull fracture, midline shift, mass effect
*Labs: CBC, coagulation studies, type and screen
*Evaluate for [[Cervical spine injury|cervical spine injury]] and other traumatic injuries


==Management==
==Management==
*ABCs — secure airway early if declining GCS
*Emergent neurosurgical consultation
*Indications for surgical evacuation (craniotomy):
**EDH >30 mL regardless of GCS
**Clot thickness >15 mm or midline shift >5 mm
**GCS declining or GCS <9 with pupil abnormalities
*ICP management while awaiting surgery:
**Elevate head of bed 30 degrees
**[[Mannitol]] 1-1.5 g/kg IV bolus or [[Hypertonic saline]] 23.4% 30 mL IV
**Controlled [[Intubation (main)|intubation]] if GCS <=8; target PaCO2 35 mmHg
**Avoid hypotension (maintain MAP >80)
*Small EDH (<30 mL, <15 mm thick, <5 mm shift, GCS >8): may be managed non-operatively with serial CT and close neuro monitoring
==Disposition==
*All EDH require admission with neurosurgical involvement
*ICU admission for any operative EDH or declining neurologic exam
*Repeat CT in 6-8 hours for non-operative cases


==See Also==
==See Also==
*[[Intracranial Hemorrhage (Main)]]
*[[Subdural hemorrhage]]
*[[Head Trauma]]
*[[Head trauma (main)]]
*[[Increased intracranial pressure]]
*[[Subarachnoid hemorrhage]]


==Source==
==References==
<references/>


[[Category:Neuro]]
[[Category:Neurology]]
[[Category:Trauma]]
[[Category:Trauma]]

Revisión actual - 09:26 22 mar 2026

Background

  • Bleeding between the skull and dura mater, typically from rupture of the middle meningeal artery
  • Usually associated with temporal bone fracture[1]
  • Accounts for 1-4% of traumatic head injuries
  • Bimodal age distribution: most common in adolescents and young adults
  • Rare in elderly (dura more adherent to skull) and infants <2 years
  • Carries ~5-10% mortality with prompt treatment; higher if uncal herniation occurs

Clinical Features

  • Classic presentation (lucid interval in ~30% of cases):
    • Initial loss of consciousness (LOC) after head trauma
    • Transient period of lucidity
    • Rapid deterioration with decreasing GCS, ipsilateral pupil dilation
  • Headache, nausea, vomiting
  • Signs of increased ICP: Cushing response (hypertension, bradycardia, irregular respirations)
  • Ipsilateral fixed, dilated pupil (uncal herniation)
  • Contralateral hemiparesis
  • May present without lucid interval, especially with severe injury

Differential Diagnosis

Evaluation

  • Non-contrast CT head — test of choice[2]
    • Biconvex (lens-shaped) hyperdense collection
    • Does NOT cross suture lines (confined by dural attachments)
    • May see associated skull fracture, midline shift, mass effect
  • Labs: CBC, coagulation studies, type and screen
  • Evaluate for cervical spine injury and other traumatic injuries

Management

  • ABCs — secure airway early if declining GCS
  • Emergent neurosurgical consultation
  • Indications for surgical evacuation (craniotomy):
    • EDH >30 mL regardless of GCS
    • Clot thickness >15 mm or midline shift >5 mm
    • GCS declining or GCS <9 with pupil abnormalities
  • ICP management while awaiting surgery:
    • Elevate head of bed 30 degrees
    • Mannitol 1-1.5 g/kg IV bolus or Hypertonic saline 23.4% 30 mL IV
    • Controlled intubation if GCS <=8; target PaCO2 35 mmHg
    • Avoid hypotension (maintain MAP >80)
  • Small EDH (<30 mL, <15 mm thick, <5 mm shift, GCS >8): may be managed non-operatively with serial CT and close neuro monitoring

Disposition

  • All EDH require admission with neurosurgical involvement
  • ICU admission for any operative EDH or declining neurologic exam
  • Repeat CT in 6-8 hours for non-operative cases

See Also

References

  1. Bullock MR, et al. Surgical management of acute epidural hematomas. Neurosurgery. 2006;58(3 Suppl):S7-15. PMID 16710967.
  2. Dent DL, et al. Prognostic factors after acute subdural hematoma. J Trauma. 1995;39(1):36-43. PMID 7636907.