Diferencia entre revisiones de «AVR ST elevation»
Sin resumen de edición |
Sin resumen de edición |
||
| Línea 1: | Línea 1: | ||
== | ==Background== | ||
*AVR elevation is commonly thought of as a sign of Left Main Coronary Artery (LMCA) occlusion. However, STE 0.5mm or greater in lead aVR to be present in 78% of patients with and 14% of patients without LMCA stenosis.<ref>Kosuge M et al. Predictors of Left Main or Three-Vessel Disease in Patients Who Have Acute Coronary Syndromes with Non-ST-Segment Elevation. Am J Cardiol 2005; 95: 1366 – 9. PMID: 15904646</ref> | *AVR elevation is commonly thought of as a sign of Left Main Coronary Artery (LMCA) occlusion. However, STE 0.5mm or greater in lead aVR to be present in 78% of patients with and 14% of patients without LMCA stenosis.<ref>Kosuge M et al. Predictors of Left Main or Three-Vessel Disease in Patients Who Have Acute Coronary Syndromes with Non-ST-Segment Elevation. Am J Cardiol 2005; 95: 1366 – 9. PMID: 15904646</ref> | ||
*Use > 1mm and the clinical status of a patient if activating the cath lab based on aVR and concern for a [[STEMI]] | *Use > 1mm and the clinical status of a patient if activating the cath lab based on aVR and concern for a [[STEMI]] | ||
==Differential Diagnosis== | ==Differential Diagnosis== | ||
Isolated elevation AVR is poorly specific for a LMCA. The following are other causes of aVR elevation | {{ST elevation DDX}} | ||
==Evaluation== | |||
Isolated elevation AVR is poorly specific for a LMCA. The following are other causes of aVR elevation: | |||
*Nontraumatic thoracic aortic dissection | *Nontraumatic thoracic aortic dissection | ||
*Massive [[Pulmonary Embolism]] | *Massive [[Pulmonary Embolism]] | ||
| Línea 9: | Línea 13: | ||
*[[Left bundle branch block]] (LBBB) | *[[Left bundle branch block]] (LBBB) | ||
*Left Ventricular Hypertrophy (LVH) with Strain Pattern | *Left Ventricular Hypertrophy (LVH) with Strain Pattern | ||
*Severe | *Severe atrial tachydysrhythmias | ||
==Management== | |||
==External Links== | ==External Links== | ||
Revisión del 14:41 6 abr 2019
Background
- AVR elevation is commonly thought of as a sign of Left Main Coronary Artery (LMCA) occlusion. However, STE 0.5mm or greater in lead aVR to be present in 78% of patients with and 14% of patients without LMCA stenosis.[1]
- Use > 1mm and the clinical status of a patient if activating the cath lab based on aVR and concern for a STEMI
Differential Diagnosis
ST Elevation
- Cardiac
- ST-segment elevation myocardial infarction (STEMI)
- Post-MI (ventricular aneurysm pattern)
- Previous MI with recurrent ischemia in same area
- Wellens' syndrome
- Coronary artery vasospasm (eg, Prinzmetal's angina)
- Coronary artery dissection
- Pericarditis
- Myocarditis
- Aortic dissection in to coronary
- Left ventricular aneurysm
- Left ventricular pseudoaneurysm
- Early repolarization
- Left bundle branch block
- Left ventricular hypertrophy (LVH)
- Myocardial tumor
- Myocardial trauma
- RV pacing (appears as Left bundle branch block)
- Brugada syndrome
- Takotsubo cardiomyopathy
- AVR ST elevation
- Other thoracic
- Metabolic
- Drugs of abuse (eg, cocaine, crack, meth)
- Hyperkalemia (only leads V1 and V2)
- Hypothermia ("Osborn J waves")
- Medications
Evaluation
Isolated elevation AVR is poorly specific for a LMCA. The following are other causes of aVR elevation:
- Nontraumatic thoracic aortic dissection
- Massive Pulmonary Embolism
- Massive GI bleed
- Left bundle branch block (LBBB)
- Left Ventricular Hypertrophy (LVH) with Strain Pattern
- Severe atrial tachydysrhythmias
Management
External Links
References
- ↑ Kosuge M et al. Predictors of Left Main or Three-Vessel Disease in Patients Who Have Acute Coronary Syndromes with Non-ST-Segment Elevation. Am J Cardiol 2005; 95: 1366 – 9. PMID: 15904646