Arteriovenous malformation - Revision history

ClaireLewis: Redirected page to Arteriovenous malformation (CNS)

2019-10-01T23:02:12Z

Redirected page to Arteriovenous malformation (CNS)

← Revisión anterior		Revisión del 23:02 1 oct 2019
Línea 1:		Línea 1:
	~~==Background==~~		#REDIRECT[[Arteriovenous malformation (CNS)]]
	*Focal abnormal conglomerations of dilated arteries and veins in the brain parenchyma
	**Arterial blood flows directly into draining veins without capillary beds creating high pressure channels
	**May rupture, causing intracerebral hemorrhage +/- intraventricular hemorrhage. Mechanism of rupture incompletely understood.
	*Prevalence 0.14%
	*Congenital lesions
	**Majority are sporadic, not familial
	**Associated with Osler- Weber-Rendu disease and Sturge-Weber syndrome
	*Lifelong risk of bleeding
	**2-4% per year, cumulative

	~~==Clinical Features==~~
	*Hemorrhage (most common)
	**42-72% of clinically apparent AVMs
	**Usually by age 20-49
	*[[Seizures]]
	*Ischemia (by vascular steal – rare)
	*[[~~Headaches]]~~

	~~==Differential Diagnosis==~~
	*Venous angioma
	*Cavernous malformation
	*Capillary telangiectasia
	~~==Evaluation==~~
	~~===Work-up===~~
	*CT/CTA
	**CT delineates acute hemorrhage, CTA to show abnormal vasculature
	*MRI/MRA
	**Vessels appear as abnormal flow voids
	**Both better than CT for visualizing structures in relation to AVM
	*Angiography
	**Gold standard to evaluate the architecture including arterial feeding, venous drainage, and AVM-associated aneurysms
	~~===Evaluation===~~

	~~==Management==~~
	*Surgical Resection
	**Traditionally treatment of choice
	*Radiosurgery (Gamma Knife or CyberKnife)
	*Embolization (~~usually an adjunct treatment~~)
	*Combination of above three in some cases
	**Decision is based on AVM size, location, and patient factors

	~~==Disposition==~~
	*In cases of acute hemorrhage or neurological decline, obvious need for immediate neurosurgical consultation
	*If patient presents with headache or seizure and lesion is then found, may simply warrant outpatient neurosurgical evaluation

	~~==See Also==~~

	~~==External Links==~~

	~~==References==~~
	~~<references/>~~

	~~[[Category:Neurology]]~~
	~~[[Category:Vascular~~]]

Neil.m.young: Text replacement - "==Diagnosis==" to "==Evaluation=="

2016-07-23T10:52:01Z

Text replacement - "==Diagnosis==" to "==Evaluation=="

← Revisión anterior		Revisión del 10:52 23 jul 2016
Línea 22:		Línea 22:
	*Cavernous malformation		*Cavernous malformation
	*Capillary telangiectasia		*Capillary telangiectasia
	==~~Diagnosis~~==		==Evaluation==
	===Work-up===		===Work-up===
	*CT/CTA		*CT/CTA

Ostermayer: Text replacement - "Category:Neuro" to "Category:Neurology"

2016-03-22T14:14:35Z

Text replacement - "Category:Neuro" to "Category:Neurology"

← Revisión anterior		Revisión del 14:14 22 mar 2016
Línea 52:		Línea 52:
	<references/>		<references/>

	[[Category:~~Neuro~~]]		[[Category:Neurology]]
	[[Category:Vascular]]		[[Category:Vascular]]

Neil.m.young at 15:00, 18 October 2015

2015-10-18T15:00:09Z

← Revisión anterior		Revisión del 15:00 18 oct 2015
Línea 53:		Línea 53:

	[[Category:Neuro]]		[[Category:Neuro]]
			[[Category:Vascular]]

Neil.m.young: format

2015-09-04T20:28:59Z

format

← Revisión anterior		Revisión del 20:28 4 sep 2015
Línea 22:		Línea 22:
	*Cavernous malformation		*Cavernous malformation
	*Capillary telangiectasia		*Capillary telangiectasia
			==Diagnosis==
	==~~Workup~~==		===Work-up===
	*CT/CTA		*CT/CTA
	**CT delineates acute hemorrhage, CTA to show abnormal vasculature		**CT delineates acute hemorrhage, CTA to show abnormal vasculature
Línea 31:		Línea 31:
	*Angiography		*Angiography
	**Gold standard to evaluate the architecture including arterial feeding, venous drainage, and AVM-associated aneurysms		**Gold standard to evaluate the architecture including arterial feeding, venous drainage, and AVM-associated aneurysms
			===Evaluation===

	==Management==		==Management==
Línea 48:		Línea 49:
	==External Links==		==External Links==

	==~~Sources~~==		==References==
	*Friedlander, Robert. Arteriovenous Malformations of the Brain. The New England Journal of Medicine. June 2007; 356: 2704-12.		<references/>
	*Greenberg, Mark. Handbook of Neurosurgery. New York: Thieme, 2006. 835-838.

	[[Category:Neuro]]		[[Category:Neuro]]

Catierey: added links

2015-09-04T16:59:55Z

added links

← Revisión anterior		Revisión del 16:59 4 sep 2015
Línea 14:		Línea 14:
	**42-72% of clinically apparent AVMs		**42-72% of clinically apparent AVMs
	**Usually by age 20-49		**Usually by age 20-49
	*Seizures		*[[Seizures]]
	*Ischemia (by vascular steal – rare)		*Ischemia (by vascular steal – rare)
	*Headaches		*[[Headaches]]

	==Differential Diagnosis==		==Differential Diagnosis==

Rossdonaldson1: /* Sources */

2014-12-02T16:15:07Z

Sources

← Revisión anterior		Revisión del 16:15 2 dic 2014
Línea 51:		Línea 51:
	*Friedlander, Robert. Arteriovenous Malformations of the Brain. The New England Journal of Medicine. June 2007; 356: 2704-12.		*Friedlander, Robert. Arteriovenous Malformations of the Brain. The New England Journal of Medicine. June 2007; 356: 2704-12.
	*Greenberg, Mark. Handbook of Neurosurgery. New York: Thieme, 2006. 835-838.		*Greenberg, Mark. Handbook of Neurosurgery. New York: Thieme, 2006. 835-838.

			[[Category:Neuro]]

Leilanicaraballo: Created page with "==Background== *Focal abnormal conglomerations of dilated arteries and veins in the brain parenchyma **Arterial blood flows directly into draining veins without capillary beds..."

2014-10-31T02:38:48Z

Created page with "==Background== *Focal abnormal conglomerations of dilated arteries and veins in the brain parenchyma **Arterial blood flows directly into draining veins without capillary beds..."

Página nueva

==Background==
*Focal abnormal conglomerations of dilated arteries and veins in the brain parenchyma
**Arterial blood flows directly into draining veins without capillary beds creating high pressure channels
**May rupture, causing intracerebral hemorrhage +/- intraventricular hemorrhage. Mechanism of rupture incompletely understood.
*Prevalence 0.14%
*Congenital lesions
**Majority are sporadic, not familial
**Associated with Osler- Weber-Rendu disease and Sturge-Weber syndrome
*Lifelong risk of bleeding
**2-4% per year, cumulative

==Clinical Features==
*Hemorrhage (most common)
**42-72% of clinically apparent AVMs
**Usually by age 20-49
*Seizures
*Ischemia (by vascular steal – rare)
*Headaches

==Differential Diagnosis==
*Venous angioma
*Cavernous malformation
*Capillary telangiectasia

==Workup==
*CT/CTA
**CT delineates acute hemorrhage, CTA to show abnormal vasculature
*MRI/MRA
**Vessels appear as abnormal flow voids
**Both better than CT for visualizing structures in relation to AVM
*Angiography
**Gold standard to evaluate the architecture including arterial feeding, venous drainage, and AVM-associated aneurysms

==Management==
*Surgical Resection
**Traditionally treatment of choice
*Radiosurgery (Gamma Knife or CyberKnife)
*Embolization (usually an adjunct treatment)
*Combination of above three in some cases
**Decision is based on AVM size, location, and patient factors

==Disposition==
*In cases of acute hemorrhage or neurological decline, obvious need for immediate neurosurgical consultation
*If patient presents with headache or seizure and lesion is then found, may simply warrant outpatient neurosurgical evaluation

==See Also==

==External Links==

==Sources==
*Friedlander, Robert. Arteriovenous Malformations of the Brain. The New England Journal of Medicine. June 2007; 356: 2704-12.
*Greenberg, Mark. Handbook of Neurosurgery. New York: Thieme, 2006. 835-838.